As aging progresses, the human body brings weakening cardiovascular function, metabolism, nervous system, and muscular system, which leads to decreased vascular elasticity and increased blood viscosity, which leads to cardiovasculase (CVD) or cerebral infarction.
Cardiovascular disease is largely related to living habits such as smoking, drinking, sitting down, and eating habits, especially with eating habits.
In addition, in recent 10 years, high blood pressure and heart disease have been on the rise, preventing and treating cardiovascular diseases.
The importance of it is emerging.
Cardiovascular disease is largely caused by hypertension, arteriosclerosis, strokes, and myocardial infarction.
Risk factors for cardiovascular diseases include age, family history, smoking, sedentary life, high blood pressure, obesity, and hyperlipidemia, and the more risk factors, the higher the likelihood of cardiovascular disease.
In particular, older adults are more likely to be overweight and obese due to decreased activity along with high fat eating habits, which can lead to cardiovascular and coronary heart disease (CHD) such as hypertension, arteriosclerosis and myocardial infarction.
In adults over 65 years of age, women have a higher mortality rate from hypertension, ischemic heart disease, cerebrovascular disease and other heart disease than men.
This is because women suffer from natural or surgical menopause as they age, which increases the risk of high blood pressure and cardiovascular disease due to reduced secretion of estrogen, a female hormone.
According to a prior study, improvements in living habits such as eating and exercising habits were greatly effective in preventing and treating cardiovascular diseases.
Degradation of the cardiovascular system due to aging is reported to show decreased vascular elasticity and static correlation.
Decreasing vascular elasticity reduces the buffer function of the arteries, which increases the pulse pressure, aortic disability, and the load of the heart, causing left ventricular hypertrophy and cardiovascular diseases such as high blood pressure and arteriosclerosis.
However, the increase in physical activity can prevent this decrease in vascular elasticity.
The long-term gateball exercise gave a positive change to the vascular elasticity of the elderly, and 12 weeks of medium-intensity exercise training for elderly women reported improved vascular elasticity.
Continuous exercise has been shown to increase the concentration of nitricoxides synthase (NOS), the production of nitric oxide (NO) by NOS activates the concentration of guanylate cyclase and increases the concentration of cGMP, relaxing the blood vessels and reducing the stiffening of ca2+ in cells.
In addition, high concentrations of n-3 PUFA intake, such as fishoil, DHA, and EPA, have been reported to have reduced blood pressure.
Consumption of high concentrations of n-3PUFAs improves blood pressure, arterial elasticity, lipid and immune systems, and reduces heart rate (HR) at rest.
It has been reported that ingestion of n-3PUFAs over three months improves lipid quality in patients with Pulmonary Arterial Diseases (PAD), improves endothelial function, and reduces ischemia.
A study of young adults showed no change in the group that consumed placebo, while the group that consumed DHA+EPA showed a significant decrease in blood pressure when stable.
In addition, fish-based eating habits reduce the incidence of sudden death, cardiovascular disease, and coronary heart disease, so high concentrations of n-3PUFAs are expected to improve the quality of life in older women by reducing cardiovascular risk and mortality.
In addition, the intake of n-3PUFAs affects cardiovascular reactions during exercise, improving the endothelial function of the peripheralvasculature.
During exercise, cardiac output (CO), myocardial contraction, blood pressure, and heart rate increases due to the activation of the sympathetic nervous system, and increases blood flow by expanding blood vessels in the skeletal muscle and heart.
In a study by Walser & Stebins, six weeks of DHA+EPA intake in young adults reduced mean arterial blood pressure (MAP) when stabilized, increased stroke (SV) and cardiac output, and increased vas-systemic resistance during moderate intensity Resistance: SVR) has been reported to be reduced, and these results suggest that ingestion of n-3 PUFAs reduces the burden on the heart and blood vessels during exercise and increases blood flow to skeletal muscles.
These results show that long-term intake of n-3 PUFAs mitigates excessive blood pressure increases during exercise, furthermore, the stomach of cardiovascular disease.
It is thought to reduce the risk.
Numerous studies have been conducted on the effects of ingestion of n-3PUFAs on cardiovascular reactions, with recent assumptions suggesting that ingestion of n-3PUFAs will affect the sympathetic nervous system when stable and exercising.
Norepinephrine (NE) is a neurotransmitter that can examine sympathetic nervous system activity, which increases heart rate and cardiac retraction, and acts with a smooth muscle ﬁ-receptor to constrict blood vessels.
A prior study of norepinephrine found that nine weeks of EPA+DHA intake reported a decrease in concentration of norepinephrine in adult men and women, while six weeks of EPA administration in STZ-induced diabetic rats caused a decrease in heart concentration.
As such, many studies have been conducted on the effectiveness of n-3PUFAs, but there is a very lack of research on the effects of n-3PUFAs on cardiovascular reactions during stabilization and exercise in older women.
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